Proponents of intelligent design would agree that biology is imperfect and biomedicine should be encouraged to cure diseases and fix problems.
For my part, I think it’s better to approach the data without assumptions and to let the evidence speak for itself.
Because of the “dysmorphic” phenotype from a homozygous genotype, these mutant genes would be highly unlikely to become fixed in a population.
The interpretation of the results as showing “latent” genetic capabilities has teleological overtones that are compatible with intelligent design.
I appreciate that Dr. Falk and his colleagues at BioLogos would like to engage in a productive “dialogue” with proponents of intelligent design.